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Can depleting Staufen1 in cells of humans suffering from ataxia or amyotrophic lateral sclerosis (ALS) improve their symptoms?

Wow,

September 7, 2018
Source:
University of Utah
Summary:
Scientists report for the first time that a protein, called Staufen1, accumulates in cells of patients suffering from degenerative ataxia or amyotrophic lateral sclerosis (ALS), also known as Lou Gehrig's disease. Depleting the protein from affected mice improved symptoms including motor function.
https://www.sciencedaily.com/releases/2018/09/180907135917.htm

67 day(s) ago

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Answers (1)

Kravenhead
Humans don't always respond as mice do to drugs, but this sounds pretty good so far.
If further research produces the same results in humans, that'll be wonderful.
It will open up new avenues for developing treatments.
A few years ago I would have said... Slowly but surely, they're making headway. But today it's more like fast and furiously.

Posted 67 day(s) ago

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Question Title Can depleting Staufen1 in cells of humans suffering from ataxia or amyotrophic lateral sclerosis (ALS) improve their symptoms?
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